Quantitative metabolomics services for biomarker discovery and validation.
Specializing in ready to use metabolomics kits.
Your source for quantitative metabolomics technologies and bioinformatics.
Loader

Loading Pathway...

Antithrombin- III Coagulation factor X Prothrombin Thrombin Fibrinogen alpha chain Fibrinogen beta chain Fibrinogen gamma chain Fibrin (loose) Fibrin (loose) Fibrin (loose) Sulodexide Calcium Fibrin (mesh) Fibrin (mesh) Fibrin (mesh) The inhibition of factor Xa halts the production of thrombin from prothrombin. Since fibrin is formed from fibrinogen with the help of thrombin, its production is also ceased Sulodexide is administered intravenously directly into the bloodstream Antithrombin, potentiated by heparin, inhibits coagulation factor Xa, which causes a disruption in the normal coagulation cascade Without fibrin, platelets are unable to clump together to form clots, preventing coagulation Extracellular Space Blood Vessel Sulodexide acts to potentiate antithrombin III and further inhibit coagulation factor x
Melanosome SERPINC1 F10 Unknown Unknown Unknown Unknown FGG Unknown Unknown FGG Sulodexide Unknown Unknown FGG
SERPINC1 F10 FGG FGG Sulod Ca2+ FGG The inhibition of factor Xa halts the production of thrombin from prothrombin. Since fibrin is formed from fibrinogen with the help of thrombin, its production is also ceased Sulodexide is administered intravenously directly into the bloodstream Antithrombin, potentiated by heparin, inhibits coagulation factor Xa, which causes a disruption in the normal coagulation cascade Without fibrin, platelets are unable to clump together to form clots, preventing coagulation Extracellular Space Blood Vessel Sulodexide acts to potentiate antithrombin III and further inhibit coagulation factor x
Melanosome SERPINC1 F10 FGG FGG Sulod FGG